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There is ample evidence that dietary and endogenous glutamate (directly or indirectly) influences glutamatergic neurotransmission in the gastrointestinal tract and the brain, respectively

Mitochondrial translational defect extends lifespan in C

Complementing these effects, KPV exerts tissue-selective anti-inflammatory activity by inhibiting NF-B signaling, reducing adhesion molecule expression, and suppressing pro-inflammatory cytokines, thereby maintaining a controlled immune environment conducive to repair

Conclusion Emerging evidence underscores the cardioprotective effects of GLP-1RAs, which make them potentially useful for different subpopulations of patients with cardiovascular diseases, including those with HFpEF